Paradigm Home
NK Cell's(TitlePage scanned)
NK Cell Modulation ?
Ref. 4 NK Cell Modulation (scanned)
Title Page scan (it can be done, but how?)
EMF Therapy Treatment Articles
Magnetic Water Treatment (As used in Industry,Physics)
Under Construction.
Other articles/abstract's to be posted.
Other abstract thoughts.
Some abstract thoughts.
In a boiler or cooling tower, calcium based scale is controlled
by adjusting the pH and
magnetic water treatment
While the human body must be subject to the physics involved,
the process is further complicated by the expression of calcium
from natural killer cells. As these articles demonstrate, fatty
acids can help to modulate this. But for calcium to be processed
properly, the iodine, magnesium and other minerals MUST be
balanced in there ratio. ( As concrete has a ratio to it's
ingredients).
Unfortunately, I do not have a copy of a study showing how this
"ratio" can effect blood pressure and the circulatory system. But
if my memory serves me correctly, they overlooked the relative
variables of the pH and essential fatty acids. And there are no doubt
other's, as yet unidentified (Strontium 90,+ *.* ?).
Of which translates in to the further study study being needed.
Being that calcium is the main transporter of intercellular signals,
the manner in which it precipitates out of a solution and can be
re-entrained into a solution should be a factor in signal distortion.
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MEDLINE: Biomedicine, 1990- Sun Aug 23 21:28:18 1998
Document 13
Accession No.: 98287508.
Author: Kruger-M-C. Horrobin-D-F.
Title: Calcium metabolism, osteoporosis and essential fatty
acids: a review.
Source: Prog-Lipid-Res. 1997 Sep. 36(2-3). P 131-51.
Journal Title: PROGRESS IN LIPID RESEARCH.
Abstract:
Essential fatty acid (EFA)-deficient animals develop severe osteoporosis
coupled with increased renal and arterial calcification. This picture is
similar to that seen in osteoporosis in the elderly, where the loss of bone
calcium is associated with ectopic calcification of other tissues,
particularly the arteries and the kidneys. Recent mortality studies
indicate that the ectopic calcification may be considerably more dangerous
than the osteoporosis itself, since the great majority of excess deaths
in women with osteoporosis are vascular and unrelated to fractures or other
bone abnormalities. EFAs have now been shown to increase calcium absorption
from the gut, in part by enhancing the effects of vitamin D, to reduce
urinary excretion of calcium, to increase calcium deposition in bone and
improve bone strength and to enhance the synthesis of bone collagen. These
desirable actions are associated with reduced ectopic calcification. The
interaction between EFA and calcium metabolism deserves further investigation
since it may offer novel approaches to osteoporosis and also to the ectopic
calcification associated with osteoporosis which seems to be responsible for
so many deaths.
Holdings: Health Sciences Serials
SHELVED BY TITLE: Progress in lipid research
CALL NUMBER: W1 PR67L
LIB HAS: v.17:n.1-3(1978/1979), v.18(1979)-v.34(1995)
Document 14
Accession No.: 98254978.
Author: Baggio-B. Plebani-M. Gambaro-G.
Title: Pathogenesis of idiopathic calcium nephrolithiasis:
update 1997.
Source: Crit-Rev-Clin-Lab-Sci. 1998 Apr. 35(2). P 153-87.
Journal Title: CRITICAL REVIEWS IN CLINICAL LABORATORY SCIENCES.
Abstract:
Idiopathic calcium nephrolithiasis (ICN) is a frequent disease in
Western countries. The physicochemical theory of lithogenesis, which
explains stone formation by the precipitation, growth, and crystalline
aggregation of lithogenic salts in the urine, has contributed greatly to
the understanding of the pathogenesis of calcium urolithiasis. However,
several aspects are still unexplained; the co-existence of familial
occurrence, primary tubular dysfunctions with ICN, and anomalies in the
systemic handling of oxalate and calcium led to the development of a
cellular hypothesis of ICN. A number of cellular defects in the handling
of ions has been reported that involves both anion and cation transport.
"crystal grouth inhibition mechanism",pg.4 .
That Whole Report on Magnetic Water Treatment
These anomalies are probably the expression of a still unknown cellular
defect in idiopathic calcium stone formers. We suggested that an anomaly
in the cell membrane composition might be responsible for the complex
array of cell ion flux abnormalities observed in ICN. Recently,
a disorder in the n-6 polyunsaturated fatty acid series has been
described; it is characterized by a lower linoleic acid content
and a higher arachidonic acid concentration in both plasma and
erythrocyte membrane phospholipids of renal calcium stone patients.
This anomaly could cause an increased activity of ion carriers;
furthermore, it may lead to increased prostaglandin synthesis and to
secondary phenomena at the kidney, skeletal, and intestinal level. As a
consequence, critical conditions for lithogenesis in the kidney may
ensue. The data suggest a common pathogenesis for hypercalciuria
and hyperoxaluria. The systemic defect in the phospholipid arachidonic
acid level may be both of dietary or genetic origin; experimental data
suggest that the increase in delta-6 desaturase activity, the limiting
enzyme in the metabolic pathway of polyunsaturated fatty acids, might be
relevant to the pathogenesis of lipid abnormalities observed in
nephrolithiasis and to the pathogenesis of ICN and its
related problems (at the kidney, intestinal, and bone level).
Holdings: Health Sciences Serials
SHELVED BY TITLE: Critical reviews in clinical
laboratory
sciences
CALL NUMBER: W1 C555B
LIB HAS: v.12:3-6(1980) v.13(1981)--
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