~~Gout~~
| Characterized | Elevated levels of uric acid in the blood |
| Cause | -unknown -perhaps, a Diet rich in alcohol and proteins |
| Symptoms | -Acute attacks in joints |
| Diagnosis | -Blood tests -Joint fluid sample |
| Treatment | -rest for limbs -anti-inflammatory drugs -drugs to inhibit production of uric acid |
"Gout, complex disease of uncertain origin caused by the faulty metabolism of uric acid produced in the body by breakdown of protein, and resulting in elevated levels of uric acid in the blood. A diet rich in malt liquors, wines, and certain types of protein may precipitate individual attacks but does not cause the disease"
"Gout". Microsoft Encarta. © 1993-2003 Microsoft Corporation. All rights
reserved.
"Repeated attacks may result in the development of a condition known as
chronic tophaceous gout. In this condition crystals of uric acid lodge as white,
chalky material in soft body tissues and in and about the joints, where they may
cause bursitis and destruction of bone. Large and deforming deposits may, after
many years, settle in the outer margins of the ears, a characteristic feature of
the disease. Chronic gout may also cause kidney damage by the formation of uric
acid stones, a condition called urate, or gouty, nephropathy.
The treatment of both types of gout calls for complete rest of the limb and a
simple diet, low in poultry, legumes, liver and other organ meats and high water
intake to reduce the uric acid content of the body. Sodium salicylate and
corticosteroids may be prescribed, or a corticosteriod may be injected into the
affected joint. The acute phase is managed with anti-inflammatory drugs, such as
colchicine or indomethacin. Chronic gout is usually treated by agents that
promote excretion of uric acid, such as probenecid, and agents that inhibit
production of uric acid, such as allopurinol."
"Gout". Microsoft Encarta. © 1993-2003 Microsoft Corporation. All rights
reserved.
